Table 1

Summary of key information on protective immunity in bovine genital campylobacteriosis and trichomoniasis.

Characteristic
Campylobacteriosis
Trichomoniasis

Etiologic Agent
C. fetus (Gram negative bacteria)
T. foetus (Protozoa)
Parasitic Type
Extracellular noninvasive
Extracellular noninvasive
Passive Protection by Antibody
Reference 1
ND*
Specificity of Protective Antibody
Heat labile surface antigen (S-Layer – Reference 33)
Lipophosphoglycan surface antigen
Predominant Ig Class of Protective Antibody
Uterus IgG1 or IgG2 Vagina IgA or IgG1
Uterus IgG1 Vagina IgA or IgG1
Most Protective Immunization
Systemic
Systemic or Systemic priming and local boosting
Cure of Infection by Vaccination
Male and Female
Male (Female?)
Evasion of Host Responses
Antigenic Variation
Proteinase cleavage of Ig Nonspecific binding of Ig
Vaccines Available Commercially
Whole killed cells Systemic route
Whole killed cells Systemic route

*ND = Not Done

Corbeil et al. Reproductive Biology and Endocrinology 2003 1:118   doi:10.1186/1477-7827-1-118